Hypothalamic Integration of Energy Metabolism, Proceedings by A. Kalsbeek, E. Fliers, M.A. Hofman, D.F. Swaab, E.J.W. van

By A. Kalsbeek, E. Fliers, M.A. Hofman, D.F. Swaab, E.J.W. van Someren and R.M. Buijs (Eds.)

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Extra resources for Hypothalamic Integration of Energy Metabolism, Proceedings of the 24th International Summer School of Brain Research, held at the Royal Netherlands Academy of Arts and Sciences

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On the other hand, when patients or animals 16 Fig. 9. Depression; schematic illustration of an impaired interaction between the decreased activity of vasopressin neurons (AVP) in the suprachiasmatic nucleus (SCN) and the increased activity of corticotropin-releasing hormone (CRH) neurons in the paraventricular nucleus (PVN). The hypothalamo– pituitary–adrenal (HPA) system is activated in depression and affects mood, via CRH and cortisol. We found a decreased amount of vasopressin (AVP) mRNA of the SCN in depression.

1999) and by the observation that metyrapone, an inhibitor of the cortisol production, successfully treats depression in Cushing patients (Checkley, 1996). However, it should be noted that, also after correction of hypercortisolism in Cushing’s syndrome, atypical depression frequently continues to be present. , 1997). Depression with atypical symptoms in women is significantly more likely to be associated with overweight than depression with typical symptoms. , 2004). 0% vs. , 2005). , 2005).

Note that the paralysis is not instantly complete; the patient is able to reach out his arms to break the fall. Below: cataplectic attack in a hypocretin-receptor-2 mutated Doberman pincher. The attack was triggered by the excitement from receiving a piece of palatable food. , 2002; with permission). Fig. 15. Examples of staining of hypocretin-IR cell bodies in the lateral hypothalamus of an adult control subject #02-076 (A), an adult Prader–Willi patient #91-058 (B), a control infant #97-153 (C) and a Prader–Willi infant #99-079 (D).

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